Astrocyte signaling controls spike timing–dependent depression at neocortical synapses
By Rogier Min, Thomas Nevian. 2012
346 citations recorded by the source corpus.
Subfields: Literature and Literary Theory
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Citations in the corpus
- Physiology of Astroglia. (2018, neutral): “Astroglial release of glutamate, for example, has been shown to result in 1) inhibition of evoked and spontaneous excitatory or inhibitory postsynaptic currents (EPSCs or IPSCs) in hippocampus (57, 992); 2) potentiation of spontaneous excitatory postsynaptic currents (EPSCs) or inhibitory postsynapt”
- The spike timing dependence of plasticity (2012, background): “The eCB signal then diffuses retrogradely to reduce release probability either by activating CB1Rs on presynaptic terminals (Bender et al., 2006b) or by activating CB1Rs on astrocytes which in turn signal to presynaptic terminals, perhaps via preNMDARs (Min and Nevian, 2012).”
- The Convallis Rule for Unsupervised Learning in Cortical Networks (2013, background): “A substantial body of experimental evidence suggests that in juvenile neocortical neurons, the potentiation and depression components of STDP are produced by different cellular mechanisms [27–33].”
- Matching Recall and Storage in Sequence Learning with Spiking Neural Networks (2013, background): “Deterministic target sequences are of behavioral relevance, since spatiotemporal spike pattern distributions are presumably often sharply peaked in the sense that the typical spike patterns closely resemble each other.”
- Endocannabinoid signaling and synaptic function (2012, background): “Surprisingly, a recent study found that astrocytic CB1Rs were necessary and sufficient to mediate tLTD (Min and Nevian, 2012).”