The Brain Metabolite Kynurenic Acid Inhibits α7 Nicotinic Receptor Activity and Increases Non-α7 Nicotinic Receptor Expression: Physiopathological Implications

By C. Hilmas, E. Pereira, M. Alkondon, A. Rassoulpour, R. Schwarcz, E. Albuquerque. Journal of Neuroscience, 2001

764 citations recorded by the source corpus.

Subfields: Biological Psychiatry, Molecular Biology, Social Psychology

View Paper: https://www.semanticscholar.org/paper/9cd49aa7b27d8f14bbfeb660c8a19da409ad0678

How researchers see this paper

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In Molecular Biology

1. E. Pereira
2. E. Albuquerque
3. S. Rogers
4. M. Alkondon

Citations in the corpus

• Mammalian Nicotinic Acetylcholine Receptors: From Structure to Function (2009, background): “The previous report that 7-chloro-KYNA does not inhibit 7 nAChRs (210) suggests that the carboxyl group contributes to interactions of KYNA with specific residues in the APL-binding region.”
• Autism‐like behavioral phenotypes in BTBR T+tf/J mice (2008, background): “Kynurenate, a glutamate and nicotinic receptor antagonist, may play a role in neuroprotection, dendritic spine formation and dopamine release (Alkondon et al. 2004; Hilmas et al. 2001; Sapko et al. 2006; Wu et al. 2007; Yu et al. 2004).”