Pathogenesis and genetics of pre-eclampsia.
By Jm Roberts, D. Cooper. The Lancet, 2001
1,377 citations recorded by the source corpus.
Subfields: Immunology, Obstetrics and Gynecology, Pediatrics, Perinatology and Child Health
View Paper: https://www.semanticscholar.org/paper/9aa5d15d239bc64590b2c94ea67f9d4c89eb8e42
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Citations in the corpus
- The Effects of a Smoking Cessation Intervention on 14.5-Year Mortality (2005, background): “37,38 Preeclampsia has been associated with maternal pro-thrombotic disorders, thrombotic le-sionsintheplacenta,andamaternalhis-toryofthromboembolism.”
- The effects of oxidative stress on female reproduction: a review (2012, background): “The OS of preeclampsia is thought to originate from insufficient spiral artery conversion [150,188,189] which leads to discontinuous placental perfusion and a low-level ischemia-reperfusion injury [185,190,191].”
- Interactions between trophoblast cells and the maternal and fetal circulation in the mouse placenta. (2002, background): “ter (Roberts and Cooper, 2001).”
- Cardiovascular sequelae of preeclampsia/eclampsia: a systematic review and meta-analyses. (2008, background): “It is defined as the onset of hypertension and proteinuria after 20 weeks gestation.(1) Eclampsia, the concomitant development of seizures, is a rare complication of preeclampsia in the developed world (with an incidence of 1 in”
- Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia. (2003, neutral): “Preeclampsia was defined by (1) systolic blood pressure of more than 140 mmHg and diastolic blood pressure of more than 90 mmHg after 20 weeks’ gestation in a previously normotensive patient (2), newonset proteinuria (>300 mg of protein in a 24-hour urine collection or a random urine protein/creatin”