Vascular proliferation and atherosclerosis: New perspectives and therapeutic strategies

By V. Dzau, R. Braun-Dullaeus, D. Sedding. Nature Medicine, 2002

865 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Molecular Biology, Surgery

View Paper: https://www.semanticscholar.org/paper/97ee0d8f03e3dfe23d0fde003801e564c00f7271

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In Molecular Biology

1. M. Palacín
2. Antonio Zorzano
3. M. Liesa

Citations in the corpus

• Mitochondrial dynamics in mammalian health and disease. (2009, result): “This analysis is consistent with the observation that dominant-negative Ras expression inhibits VSMC proliferation in vivo (321) and that approaches that inhibit the entry of VSMCs into the cell cycle prevent restenosis secondary to the implantation of mechanical stents in patients with chronic coro”
• The role of shear stress in the pathogenesis of atherosclerosis (2005, background): “These cells contribute to intimal hyperplasia that forms the bulk of the plaque, to expression of growth factors, cytokines, and proinflammatory mediators such as monocyte chemoattractant protein-1 (MCP-1) and vascular cell adhesion molecule (VCAM) and to synthesis of matrix molecules some of which ”