Neuroanatomic observations of the brain in autism: a review and future directions

By Margaret L. Bauman, Thomas L. Kemper. International Journal of Developmental Neuroscience, 2005

906 citations recorded by the source corpus.

Subfields: Cognitive Neuroscience, Genetics, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/977489964d6e33fb307735561b45643cd8ba3330

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Citations in the corpus

The Changing Epidemiology of Autism Spectrum Disorders (2016, background): “Multiple lines of evidence suggest the etiology of ASD has prenatal origins (10).”
Evidence for topographic organization in the cerebellum of motor control versus cognitive and affective processing (2010, background): “These posterior vermal regions are also among the brain areas showing structural differences in autism spectrum disorders (Courchesne et al., 1988; Bauman and Kemper, 2005; Penn, 2006).”
Pten regulates neuronal arborization and social interaction in mice. (2006, neutral): “There have also been reports in autism cases of enlarged or abnormally oriented neurons, densely packed neuronal regions and isolated regions of atrophic neurons with reduced dendritic arborization (Bauman and Kemper, 2005; Murcia et al., 2005).”
Practice parameter for the assessment and treatment of children and adolescents with autism spectrum disorder. (2014, background): “13,24,25 The number of areas affected by autism suggests that a diverse and widely distributed set of neural systems must be affected.”
Autistic-like behavior and cerebellar dysfunction in Purkinje cell Tsc1 mutant mice (2012, neutral): “One of the most consistent pathologic findings in post-mortem studies of ASD patients is reduced cerebellar PC numbers 2 .”
Consensus Paper: Pathological Role of the Cerebellum in Autism (2012, background): “Other brain regions also display abnormalities generally resulting from reduced programmed cell death and/or increased cell proliferation, altered cell migration, and abnormal cell differentiation with reduced neuronal size and abnormal wiring [118, 230].”
Social cognition and the cerebellum: A meta-analysis of over 350 fMRI studies (2014, background): “…has been found to be dysfunctional in a broad range of psychiatric disorders such as autistic spectrum disorders, depression, and schizophrenia (Bauman and Kemper, 2005; Penn, 2006), and this finding, coupled with the presentmeta-analytic findings,may open interesting avenues for future…”
Mapping early brain development in autism. (2007, neutral): “The following is a brief summary of that literature; for other reviews, see Bauman and Kemper, 2005; Cody et al., 2002; Courch-esne et al., 2004; Courchesne and Pierce, 2005a; Herbert, 2005; Palmen et al., 2004.”
Why Are Autism Spectrum Conditions More Prevalent in Males? (2011, background): “ASC is neurobiological, evidenced by atypical brain development in structure and function [4].”
Early behavioral intervention, brain plasticity, and the prevention of autism spectrum disorder (2008, background): “Autopsy studies of ASD (Pickett & London, 2005) have documented cellular abnormalities of the amygdala including reduced numbers of neurons (Schumann & Amaral, 2006), or reduced cell size and increased neuronal cell packing density (Bauman & Kemper, 1985, 2005).”
Functional topography in the human cerebellum: A meta-analysis of neuroimaging studies (2009, neutral): “These posterior vermal regions are among the brain areas that show significant structural differences in autistic spectrum disorders (Bauman and Kemper, 2005; Courchesne et al., 1988; Penn, 2006).”
The epidemiology of autism spectrum disorders. (2007, neutral): “ASDs are heritable, but the model of inheritance”