Replication of putative candidate-gene associations with rheumatoid arthritis in >4,000 samples from North America and Sweden: association of susceptibility with PTPN22, CTLA4, and PADI4.

By Robert M. Plenge, L. Padyukov, E. Remmers, Shaun Purcell, Annette Lee, Elizabeth W. Karlson, F. Wolfe, Daniel L. Kastner, L. Alfredsson, David Altshuler and 3 more. American Journal of Human Genetics, 2005

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A Bayesian measure of the probability of false discovery in genetic epidemiology studies. (2007, neutral): “E[C(δ, H)] = C(δ, H0) Pr(H0|y) + C(δ, H1) Pr(H1|y) (18)”
Accounting for linkage disequilibrium in genome-wide association studies: A penalized regression method (2013, background): “Genetic factors underlying RA have been mapped to the HLA region on 6p21 [12], PTPN22 locus at 1p13 [2], and CTLA4 locus at 2q33 [13].”
A Genome-Wide Association Study of Psoriasis and Psoriatic Arthritis Identifies New Disease Loci (2008, methodology): “To investigate other biases[20] that could be introduced with shared controls we assessed the potential effect of substructure with the genomic-control method [21] and with EIGENSTRAT [23].”