Possible involvement of the M2 anti‐inflammatory macrophage phenotype in growth of human gliomas

By Y. Komohara, Koji Ohnishi, J. Kuratsu, M. Takeya. Journal of Pathology, 2008

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The tumor microenvironment underlies acquired resistance to CSF1R inhibition in gliomas (2016, background): “In GBM, tumorassociated macrophages and microglia (TAMs) comprise up to 30% of the bulk tumor mass (4).”
Understanding the Mysterious M2 Macrophage through Activation Markers and Effector Mechanisms (2015, background): “CD163 expression is increased in TAMs [93] and in peritonitis [83], and it is secreted into the blood in severe inflammation [94].”
Alternatively activated microglia and macrophages in the central nervous system. (2015, neutral): “…1045 1046 1047 1048 1049 1050 1051 1052 1053 1054 1055 1056 1057 1058 1059 1060 1061 1062 1063 1064 1065 1066 R. Franco, D. Fernández-Suárez / Progress in Neurobiology xxx (2015) xxx–xxx12 rrelates with the proportion of M2 cells and proliferation of ioblastoma cells (Komohara and Ohnishi, 2008).”
The microenvironmental landscape of brain tumors (2017, neutral): “(Konstantinou et al., 2014; Margolin et al., 2012; Queirolo et al., 2014).”
Immune cell infiltration as an indicator of the immune microenvironment of pancreatic cancer (2013, result): “…some studies have obtained opposite results (Forssell et al, 2007), several studies have reported that a high amount of tumour-infiltrating macrophages is correlated with a poor outcome in various cancers (Komohara et al, 2008; Shabo et al, 2008) containing pancreas cancer (Kurahara et al, 2011).”