The role of nitric oxide on endothelial function.
By D. Tousoulis, A. Kampoli, C. Tentolouris, N. Papageorgiou, Christodoulos Stefanadis. Current Vascular Pharmacology, 2012
967 citations recorded by the source corpus.
Subfields: Biochemistry, Physiology
View Paper: https://www.semanticscholar.org/paper/94a3a4d5ebd15abb3758ec6c9355dfb8f66ecdaf
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In Physiology
Citations in the corpus
- The Impact of High-Intensity Interval Training Versus Moderate-Intensity Continuous Training on Vascular Function: a Systematic Review and Meta-Analysis (2015, background): “This is because NO is not only a potent vasodilator, but also functions to prevent smooth muscle cell proliferation, and to inhibit production of pro-inflammatory factors and adhesion molecules [5].”
- Activation of Nrf2/HO-1 signaling: An important molecular mechanism of herbal medicine in the treatment of atherosclerosis via the protection of vascular endothelial cells from oxidative stress (2021, background): “NO, an important vasodilator, is important for maintaining vasodilation and systole [41].”
- Superoxide dismutases: Dual roles in controlling ROS damage and regulating ROS signaling (2018, neutral): “The endothelium-derived NO• controls the extent of vascular smooth muscle relaxation, inhibits platelet aggregation, and attenuates neutrophil adherence to the endothelium (Tousoulis et al., 2012).”
- Potential Adverse Effects of Resveratrol: A Literature Review (2020, background): “RE can also modulate NO release, which is crucial for endothelial function [123].”