Obesity‐induced inflammation: a metabolic dialogue in the language of inflammation

By A. Ferrante. Journal of Internal Medicine, 2007

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Pathophysiology of human visceral obesity: an update. (2013, background): “Chronic, low-grade inflammation caused by altered adipokine secretion may alter glucose and lipid metabolism and contribute to cardiometabolic risk of individuals with visceral obesity (149, 571).”
Oxidative Stress in Obesity: A Critical Component in Human Diseases (2014, background): “(TNF-α, IL-6 and IL-1), and neuropeptides (α-melanocyte-stimulating hormone and neuropeptide Y) have also been reported as further links between adipose tissue, OS and hypertension [112].”
Cellular and Molecular Players in Adipose Tissue Inflammation in the Development of Obesity-induced Insulin Resistance (2013, neutral): “Hence, it is now generally accepted that tissue-resident immune cells play a major role in the regulation of obesity-induced inflammation and insulin resistance, like they do in classical immunity inflammation [40].”
Obesity, Inflammation, Toll-Like Receptor 4 and Fatty Acids (2018, background): “This process is regulated by colony stimulating factors, such as macrophage-specific growth factor, called colony stimulating factor 1 (CSF-1) or macrophage colony-stimulating factor (M-CSF) [36].”