Determination of cell types and numbers during cardiac development in the neonatal and adult rat and mouse.
By Indroneal Banerjee, J. Fuseler, R. Price, T. Borg, T. Baudino. American Journal of Physiology. Heart and Circulatory Physiology, 2007
647 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Molecular Biology, Surgery
View Paper: https://www.semanticscholar.org/paper/9148051d8c236f619d32a89fe75f1270efc1b2c6
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Citations in the corpus
- The Origin of Fibroblasts and Mechanism of Cardiac Fibrosis (2010, background): “…these data show that the cardiac cellular makeup varies greatly between species and one could speculate that these differences in cellular buildup underlie differences in cardiac pressure generating capacity, cardiac collagen content, heart rate, or cardiac conductance (Banerjee et al., 2007).”
- The Pathogenesis of Cardiac Fibrosis (2013, neutral): “As the fetal circulation transitions to the neonatal circulation, elevated left ventricular pressures trigger a marked expansion of the cardiac fibroblast population within the first two neonatal weeks [15].”
- Models of cardiac tissue electrophysiology: progress, challenges and open questions. (2011, background): “The density of fibroblasts in cardiac muscle is dependent on species, age and disease (Banerjee et al., 2007, 2006).”
- Revisiting Cardiac Cellular Composition (2015, result): “Second, the use of an isolation protocol that yielded the greatest number of viable cells at the expense of cardiomyocytes may explain why flow cytometry results strongly correlated with results from immunohistochemical analyses but produced different outcomes when compared with previous studies.(2,”