Glucose tolerance in adults after prenatal exposure to famine.
By Peter D. Gluckman, C. Pinal. The Lancet, 2001
840 citations recorded by the source corpus.
Subfields: Obstetrics and Gynecology, Pediatrics, Perinatology and Child Health
View Paper: https://www.semanticscholar.org/paper/909369ca170cc0b00acbc1d541bc2f2e7c81e733
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Obstetrics and Gynecology
- Amparo Castello Climent
- D. Barker
- Frank B Hu
- Frederica Perera
- Julie Herbstman
- Rafael Doménech
- Rodrick Wallace
In Pediatrics, Perinatology and Child Health
- Xin Meng
- Amparo Castello Climent
- J. Belsky
- D. Barker
- Frank B Hu
- Nancy Qian
- Frederica Perera
- Julie Herbstman
Citations in the corpus
- Globalization of Diabetes (2011, background): “Results from the Dutch Famine Study show that adults exposed to famine conditions during World War II had increased insulin resistance than those who had not been exposed (54).”
- The Long Term Consequences of Famine on Survivors: Evidence from a Unique Natural Experiment Using China's Great Famine (2009, background): “Barker (1995) and Ravelli et al. (1998) have found that nutrition in-utero can affect health status in middle age, through its impact on chronic conditions such as coronary heart disease and diabetes, in a phenomenon widely known as the “Barker Hypothesis”.20 Poor childhood health could also…”
- Adult Consequences of Fetal Growth Restriction (2006, background): “The wartime famine in the Netherlands produced lifelong insulin resistance in babies who were in utero at the time, but with little alteration in birghtweight.(46) In babies, as in children, slowing of growth is a response to a poor environment, especially undernutrition, but body size at birth does”
- Sex and Gender Differences in Risk, Pathophysiology and Complications of Type 2 Diabetes Mellitus (2016, background): “Additionally, increased postprandial glucose levels, as well as hyperinsulinemia, were detected in adults affected by food limitation during their pregnancies (153, 154).”
- Differential susceptibility to the environment: An evolutionary–neurodevelopmental theory (2011, background): “…during the Dutch Hunger Winter of 1944–1945, who experienced a transition to food abundance in later life, and who thus acquired an enhanced risk for cardiovascular disease and early mortality (Painter et al., 2006; Ravelli et al., 1998; Ravelli, van der Meulen, Osmond, Barker, & Bleker, 1999).”
- Human Capital Inequality, Life Expectancy and Economic Growth (2008, background): “Journal compilation Royal Economic Society 2008 diseases and illnesses that an individual may suffer from.6 For example, Ravelli et al. (1998) investigate glucose tolerance in people born around the time of the famine in the Netherlands during 1944-5.”
- Prenatal environmental exposures, epigenetics, and disease (2011, positive): “Since 1992, the evidence has grown that developmental plasticity allows the fetus to make anticipatory responses to the external environment by altering the course of cellular and organ differentiation in utero in order to gain adaptive advantage for later life challenges [31,32].”