Arrhythmogenesis and Contractile Dysfunction in Heart Failure: Roles of Sodium-Calcium Exchange, Inward Rectifier Potassium Current, and Residual &bgr;-Adrenergic Responsiveness

By S. Pogwizd, Klaus Schlotthauer, Li Li, W. Yuan, Donald M. Bers. Circulation Research, 2001

811 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine

View Paper: https://www.semanticscholar.org/paper/90885352e22ca3e61f1d1b7e3226f5f43ba976f8

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In Cardiology and Cardiovascular Medicine

1. Stanley Nattel
2. Donald M. Bers

Citations in the corpus

• Arrhythmogenic ion-channel remodeling in the heart: heart failure, myocardial infarction, and atrial fibrillation. (2007, background): “(246) have shown this to be an important mechanism for the promotion of afterdepolarization-induced arrhythmias in a rabbit heart failure model.”
• Ca2+/Calmodulin–Dependent Protein Kinase Modulates Cardiac Ryanodine Receptor Phosphorylation and Sarcoplasmic Reticulum Ca2+ Leak in Heart Failure (2005, methodology): “Our aims were to assess: (1) expression of RyR2 as well as the cardiac type 2 inositol 1,4,5 trisphosphate receptor (IP3R2); (2) expression of RyR2 interacting proteins including FKBP 12.”