A metalloprotease-disintegrin participating in myoblast fusion

By Takako Yagami-Hiromasa, Tomomi Sato, T. Kurisaki, K. Kamijo, Y. Nabeshima, A. Fujisawa-Sehara. 1995

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8. Mengcheng Shen

Citations in the corpus

• A Disintegrin and Metalloproteinase (ADAM) and ADAM with Thrombospondin Motifs (ADAMTS) Family in Vascular Biology and Disease (2019, background): “ADAM-12 was initially thought to mainly take part in myogenesis during embryonic development [70].”
• Metalloprotease-Disintegrins: Links to Cell Adhesion and Cleavage of TNFα and Notch (1997, background): “Taken to-crease in the observed fusion (Yagami-Hiromasa et al., 1995).”
• The ADAMs family of metalloproteases: multidomain proteins with multiple functions. (2003, background): “ADAM12, in particular, showed a restricted spatial-temporal expression pattern that correlated well with early skeletal muscle development (Yagami-Hiromasa et al. 1995; Kurisaki et al. 1998) and muscle regeneration (Borneman et al. 2000; Galliano et al. 2000).”
• The ADAM metalloproteinases (2008, background): “As meltrin-a, ADAM-12 had originally been implicated in muscle formation, where its expression in myoblasts along with ADAM-19 (Meltrin-b) and ADAM-9 (Meltrin-c) was linked to myoblast fusion to generate multinucleated myotubes (Yagami-Hiromasa et al., 1995).”
• Essential Role for ADAM19 in Cardiovascular Morphogenesis (2004, background): “ADAM19 (also referred to as meltrin ) was initially identified in muscle cells and was later found to be expressed in several other tissues, most prominently in heart, lung, and bone (18, 27, 52), during dendritic cell differentiation (13) and Notch-induced T-cell maturation (9).”
• Essential roles of Meltrin β (ADAM19) in heart development (2004, background): “1998; Huang et al., 2003; Kurisaki et al., 2003; Peschon et al., 1998; Rooke et al., 1996; Yagami-Hiromasa et al., 1995) and are implicated in certain pathogeneses (Asakura et al., 2002; Van Eerdewegh et al., 2002).”
• ADAMs family and relatives in cardiovascular physiology and pathology. (2016, background): “ADAM12 (meltrin-alpha) was originally suggested to be involved in myogenesis during embryonic development along with ADAM9 and ADAM19 [132].”