Frontal-hippocampal double dissociation between normal aging and Alzheimer's disease.
By Denise Head, Abraham Z. Snyder, L. Girton, John C. Morris, Randy L. Buckner. 2005
161 citations recorded by the source corpus.
Subfields: Cognitive Neuroscience, Psychiatry and Mental health
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cognitive Neuroscience
- Bruce Fischl
- Anders M. Fjell
- Kristine B. Walhovd
- David H. Salat
- Douglas N. Greve
- Randy L. Buckner
- A. M. Dale
- John C. Morris
In Psychiatry and Mental health
- Randy L. Buckner
- Bruce Fischl
- John C. Morris
- Daniel S. Marcus
- Anders M. Fjell
- Kristine B. Walhovd
- John G. Csernansky
- David H. Salat
Citations in the corpus
- Human Hippocampal Neurogenesis Persists throughout Aging (2018, neutral): “, 2008), others found no volume change (Head et al., 2005), or altered hippocampal shape rather than volume (Yang et al.”
- An automated labeling system for subdividing the human cerebral cortex on MRI scans into gyral based regions of interest (2006, background): “Keywords: neurophysiology, neurophysiological resources, neuroinformatics, data intensive research, problem solving infrastructure, analysis of neurophysiological data”
- A Population-Average, Landmark- and Surface-based (PALS) atlas of human cerebral cortex (2005, unknown): “Similarly, Figure 9B shows a combined visuotopic scheme (Hadjikhani et al., 1998; Van Essen, 2004b) and orbitofrontal partitioning schemes (Öngur et al., 2003; Van Essen at al., 2005) that have been registered in this fashion.”
- What is normal in normal aging? Effects of Aging, Amyloid and Alzheimer’s Disease on the Cerebral Cortex and the Hippocampus (2014, background): “For instance, while degeneration of the medial temporo-parietal memory network is characteristic for AD, change in a frontostriatal network supporting executive functions has been proposed as a hallmark of healthy aging (Buckner, 2004; Head et al., 2005) (see Fig.”
- Memory and executive function in aging and AD: multiple factors that cause decline and reserve factors that compensate. (2004, background): “One Head et al. (2005) (see also Meguro et al., 2003) recently noted that anterior (frontal) white matter differences,source of evidence for mechanistic separation comes from rare genetic mutations.”
- Automated MRI measures identify individuals with mild cognitive impairment and Alzheimer's disease (2009, unknown): “Data from subsets of these participants have been published in previous studies (Salat et al ., 2004; Buckner et al ., 2004, 2005; Head et al ., 2005; Dickerson et al ., 2009).”
- High consistency of regional cortical thinning in aging across multiple samples. (2009, background): “According to this theory, early AD selectively attacks the medial-temporal lobes and adjacent cortical areas, leading to memory problems, whereas an anterior to posterior gradient exists in healthy aging, causing executive problems (Head et al. 2005).”
- New diagnostic EEG markers of the Alzheimer’s disease using visibility graph (2010, background): “Loss of neuronal synapses (Hampel et al. 2002; Adeli et al. 2005a), axonal degeneration, and neuronal cell body death (Fjell et al. 2010) in AD result in changes in various regions of the brain anatomically (structurally) and physiologically (functionally) (Head et al. 2005).”
- Open Access Series of Imaging Studies (OASIS): Cross-sectional MRI Data in Young, Middle Aged, Nondemented, and Demented Older Adults (2007, methodology): “Portions of the clinical, demographic, and image data obtained from subjects in this release have been used in previous studies (Buckner et al., 2004, 2005, in press; Burns et al., 2005; Fotenos et al., 2005, in press; Head, Snyder, Girton, Morris, & Buckner, 2005; Salat et al., 2004) and made pub”