Complement-like protein TEP1 is a determinant of vectorial capacity in the malaria vector Anopheles gambiae.

By S. Blandin, Shin-Hong Shiao, L. Moita, C. Janse, A. Waters, F. Kafatos, E. Levashina. Cell, 2004

610 citations recorded by the source corpus.

Subfields: Immunology, Insect Science, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/8f11c7443de3f426550afdcaae7bbec8886bacd1

How researchers see this paper

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In Immunology

1. J. Hoffmann
2. M. Strand
3. B. Lemaître
4. N. Ratcliffe
5. I. Dubovskiy
6. V. Glupov
7. N. Kryukova
8. M. J. Lehane

In Insect Science

1. J. Hoffmann
2. B. Lemaître
3. M. Strand
4. N. Ratcliffe
5. I. Dubovskiy
6. V. Glupov
7. N. Kryukova
8. M. J. Lehane

In Molecular Biology

1. M. Strand

Citations in the corpus

• The host defense of Drosophila melanogaster. (2007, background): “An opsonin function for a TEP member has been documented in Anopheles gambiae where TEP1 is also involved in parasite killing of Plasmodium (182).”
• Encapsulation and nodulation in insects (2016, background): “Interestingly, the PRRs of Anopheles gambiae against Plasmodium demonstrate similarity to those involved in bacteria recognition (Blandin et al., 2004; Dong et al., 2006; Dong and Dimopoulos, 2009; Sandiford et al., 2015).”
• The insect cellular immune response (2008, background): “In addition, hemocytes appear to be the primary source of these effector molecules (Blandin et al., 2004; Osta et al., 2004; Riehle et al., 2006).”