Fetal programming of coronary heart disease.

By D. Barker. Trends in endocrinology and metabolism, 2002

687 citations recorded by the source corpus.

Subfields: Endocrinology, Diabetes and Metabolism, Obstetrics and Gynecology, Pediatrics, Perinatology and Child Health

View Paper: https://www.semanticscholar.org/paper/8bd4820e1425244dbfd5844337aa3ab0e89494a1

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Citations in the corpus

High antenatal maternal anxiety is related to ADHD symptoms, externalizing problems, and anxiety in 8- and 9-year-olds. (2004, background): “…of development, they exercise organizational effectsFor program 1086 Van den Bergh and Marcoen some set pointsFin a variety of systems (e.g., metabolic, cardiovascular, or immune system) by different underlying mechanisms (Barker, 1998, 2002; Coe, Lubach, & Karaszweski, 1999; Nathanielsz, 1999).”
Antenatal maternal stress and long-term effects on child neurodevelopment: how and why? (2007, background): “…Publishing, 9600 Garsington Road, Oxford OX4 2DQ, UK and 350 Main Street, Malden, MA 02148, USA been shown to place individuals at risk for developing features of cardiovascular disease and metabolic disorders such as diabetes, in adulthood (Barker, 2002, 2004, 2005; Barker & Hanson, 2004).”
Predictive adaptive responses and human evolution. (2005, background): “[ 4 ]). This term, which is derived by analogy with computer software from the concept of ‘genetic programming’ [40], is unfortunate because it implies a predetermined trajectory of development insulated from any subsequent epigenetic or environmental input.”
Consciousness: A Mathematical Treatment of the Global Neuronal Workspace Model (2005, background): “Work by Barker and colleagues (Barker, 2000; Barker et al., 2002; Eriksson et al., 2000; Godfrey and Barker, 2001; Osmond and Barker, 2000) suggests that those who develop CHD grow differently from others, both in utero and during childhood. Slow growth during fetal life and infancy is followed by a”
Prenatal Maternal Anxiety and Depression Predict Negative Behavioral Reactivity in Infancy (2004, background): “The selective effects of prenatal experiences on behavioral reactivity support the hypothesis that the prenatal environment exerts programming effects on the fetus with consequences for infant behavior (Barker, 2002; Welberg & Seckl, 2001).”
A meta-analysis of pregnancy outcomes in women with polycystic ovary syndrome. (2006, background): “hyperinsulinaemia) may effect neuroendocrine systems regulating body weight, food intake and metabolism, with consequences for long-term health in the offspring ( Barker, 2002 ).”
Antenatal maternal anxiety and stress and the neurobehavioural development of the fetus and child: links and possible mechanisms. A review. (2005, result): “This finding has been reproduced in many independent studies, although it appears to be the ponderal index rather than birth weight that matters (for reviews see [9] for coronary heart disease; [10] for obesity).”
Fetal programming of hypothalamo‐pituitary‐adrenal function: prenatal stress and glucocorticoids (2006, background): “However, more recent human epidemiological evidence indicating that the fetal and early postnatal environment can influence susceptibility to later disease (Barker, 2002) has rejuvenated the field.”
The Timing of Prenatal Exposure to Maternal Cortisol and Psychosocial Stress is Associated with Human Infant Cognitive Development (2010, background): “…experimental studies with rodents that exposure to early life stress can have consequences for cognitive functioning that are not observable until adulthood (Brunson et al., 2005) as well as from human observational studies associating size at birth with adult health outcomes (Barker, 1998; 2002).”
Prenatal exposure to maternal depression and cortisol influences infant temperament. (2007, background): “Thus, the timing of the stimulus during development coupled with the timetable for organogenesis determines the nature of the programming effect (Barker, 1998, 2002).”