Onset of maternal arterial blood flow and placental oxidative stress. A possible factor in human early pregnancy failure.
By Eric Jauniaux, A. Watson, J. Hempstock, Y. Bao, J. Skepper, G. Burton. American Journal of Pathology, 2000
1,081 citations recorded by the source corpus.
Subfields: Immunology, Obstetrics and Gynecology, Pediatrics, Perinatology and Child Health
View Paper: https://www.semanticscholar.org/paper/8b535c860100e520608d1fb9a928163c5651d19a
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In Immunology
In Obstetrics and Gynecology
In Pediatrics, Perinatology and Child Health
Citations in the corpus
- The effects of oxidative stress on female reproduction: a review (2012, background): “expression and activity increases with gestational age [148].”
- Growth and function of the normal human placenta. (2004, background): “At this time, oxygen tension rises from b20 mm Hg (8 weeks) to N50 mm Hg at 12 weeks [16].”
- Placental-related diseases of pregnancy: involvement of oxidative stress and implications in human evolution (2006, background): “Consequently, other strategies need to be employed to restrict exposure of the fetus to O2, and as a result, the human placenta is exposed to major changes in O2 concentration from conception to delivery (Jauniaux et al., 2000, 2001; Burton et al., 2003).”
- Oxidative stress in the placenta (2004, background): “The reperfusion of placenta between 8 and 9 weeks may be responsible for expression of heat shock protein (HSP) 70 and formation of nitrotyrosine residues (markers for oxidative stress) in syncytiotrophoblast (Jauniaux et al. 2000).”