Calcium ion in skeletal muscle: its crucial role for muscle function, plasticity, and disease.

By M. Berchtold, H. Brinkmeier, Markus Müntener. Physiological Reviews, 2000

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Function and genetics of dystrophin and dystrophin-related proteins in muscle. (2002, background): “Calcium homeostasis is critical to many aspects of muscle function (31), and early suggestions that it might be perturbed in dystrophin-deficient muscle stemmed from several observations.”
Skeletal muscle fatigue: cellular mechanisms. (2008, background): “Most Ca(2) in the SR is bound to calsequestrin (38, 45), and the free [Ca(2) ] is 0.”
Transcriptional coactivator PGC-1α controls the energy state and contractile function of cardiac muscle (2005, background): “, exercise) by converting some glycolytic skeletal muscle fibers into more oxidative fibers (Berchtold et al., 2000).”
Regulation of Muscle Fiber Type and Running Endurance by PPARδ (2004, background): “…content and oxidative enzymes, rely on glycolytic metabolism as a major energy source, and are susceptible to fatigue, while the oxidative and contraction functions of type IIa and IIx lie between type I and IIb (Booth and Thomason 1991; Berchtold et al. 2000; Olson and Williams 2000).”
A Micropeptide Encoded by a Putative Long Non-coding RNA Regulates Muscle Performance (2015, neutral): “Ca controls the normal function of striated muscle by acting as theprimary regulator of the sarcomeric contractilemachinery and as a second messenger in the signal transduction pathways that control muscle growth, metabolism, and pathological remodeling (Bassel-Duby and Olson, 2006; Berchtold et al.”