Circuits and circuit disorders of the basal ganglia.

By M. DeLong, T. Wichmann. Archives of Neurology, 2007

1,084 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Neurology

View Paper: https://www.semanticscholar.org/paper/89ddd9a5bb3361e1bdccb722c24e6edce000265c

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In Cellular and Molecular Neuroscience

In Neurology

Citations in the corpus

Striatal plasticity and basal ganglia circuit function (2008, background): “…associated with prominent neurological disorders including Parkinson’s disease (PD) and Huntington’s disease (HD) (Albin et al., 1989; DeLong, 1990; DeLong and Wichmann, 2007; Graybiel, 2000), as well as psychiatric disorders such as obsessive-compulsive disorder (OCD) (Aouizerate et al., 2004;…”
Contributions of the basal ganglia and functionally related brain structures to motor learning. (2009, positive): “In recent years, there has been growing interest in uncovering the learning processes that span over longer time frames, as there is evidence that changes in performance can occur over multiple sessions encompassing days, weeks or even months of practice [15,16].”
Understanding dopamine and reinforcement learning: The dopamine reward prediction error hypothesis (2011, background): “These outputs pass to the dendrites of the dopamine cells, where they combine with inputs from the brainstem that likely carry signals about rewards being currently consumed (21).”
Goal-directed and habitual control in the basal ganglia: implications for Parkinson's disease (2010, background): “Many experimental investigations have shown that basal ganglia output is abnormal in patients with Parkinson's disease and in animal models of the diseas”
Subcortical processes of motor response inhibition during a stop signal task (2008, background): “Dysfunction in the circuitry involving these subcortical structures has been implicated in a number of neurological conditions, including Parkinson's disease and attention deficit hyperactivity disorder (Bevan et al., 2006; DeLong and Wichmann, 2007; Mehler-Wex et al., 2006; Robbins, 2007).”
Diagnostic and Statistical Manual of Mental Disorders (2013, background): “More specifically, because spontaneous hyperkinetic movements are believed to reflect abnormal striatal dopamine activity (DeLong and Wichmann, 2007), and dysfunction in this same circuit is also proposed to contribute to psychosis, it is possible that spontaneous dyskinesias serve as an outward…”