NGF rapidly increases membrane expression of TRPV1 heat‐gated ion channels

By Xuming Zhang, Jiehong Huang, P. McNaughton. EMBO Journal, 2005

758 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Physiology, Sensory Systems

View Paper: https://www.semanticscholar.org/paper/89cf12e85bef06e8553404874cbdaaaa00ac6ba3

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cellular and Molecular Neuroscience

1. B. Suh
2. Bertil Hille
3. L. Reichardt
4. B. Lu

In Physiology

1. B. Suh
2. Bertil Hille
3. L. Reichardt

In Sensory Systems

1. C. Dart

Citations in the corpus

• Nociceptor Sensory Neuron-Immune Interactions in Pain and Inflammation (2016, neutral): “CGRP activates MAPK pathways to induce keratinocyte proliferation and production of TNF/, IL-1b, IL-6, and NGF [100].”
• Neurotrophin-regulated signalling pathways (2006, background): “After secretion, it is captured through receptor interactions in nerve terminals, where it acts locally to regulate target innervation and nerve terminal function (Campenot 1977; Zhang et al. 2005).”
• PIP2 is a necessary cofactor for ion channel function: how and why? (2008, background): “This was followed by reports that some of the receptormediated enhancement of TRPV1 currents actually resulted from phosphorylation of the channels (97), that lipid kinase-mediated resynthesis of PIP2 is required for the TRPV1 channel recovery from desensitization (41), that PIP2 applied to excised ”