Distinct macrophage phenotypes contribute to kidney injury and repair.
By Sik Lee, Sarah C. Huen, Hitoshi Nishio, Saori Nishio, H. Lee, B. Choi, C. Ruhrberg, L. Cantley. Journal of the American Society of Nephrology, 2011
879 citations recorded by the source corpus.
Subfields: Immunology, Nephrology
View Paper: https://www.semanticscholar.org/paper/87c468ca8b04a58aeb78f5452c16b09c5917dcdc
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In Immunology
In Nephrology
Citations in the corpus
- Sequential delivery of immunomodulatory cytokines to facilitate the M1-to-M2 transition of macrophages and enhance vascularization of bone scaffolds (2014, positive): “Studies that employed fluorescent labeling showed that at least part of the M2 population that appears at later stages of wound healing are derived directly from the M1 population that presided at early stages [19,41,43].”
- Cellular and molecular mechanisms in kidney fibrosis. (2014, positive): “Since ablation of macrophages after ischemic kidney injury results in persistence of interstitial fibrosis, it is likely that reparative or M2 macrophages perform similar tasks in kidney remodeling (81, 82).”
- Macrophage phenotypes during tissue repair (2013, background): “Indeed, in mouse and human skin wounds and in injured mouse kidney, Mp expression of CD206 increases during the later stages of repair [33, 60, 61, 63].”