Detection and sequencing of Zika virus from amniotic fluid of fetuses with microcephaly in Brazil: a case study.
By G. Calvet, R. S. Aguiar, A. Melo, S. Sampaio, I. de Filippis, Allison Fabri, E. Araújo, Patricia C. de Sequeira, M. C. L. de Mendonça, L. D. de Oliveira and 8 more. Lancet. Infectious Diseases (Print), 2016
1,073 citations recorded by the source corpus.
Subfields: Epidemiology, Infectious Diseases, Public Health, Environmental and Occupational Health
View Paper: https://www.semanticscholar.org/paper/85b0dede8392afc790273bc6e53e3614f7610a98
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Citations in the corpus
- Zika Virus Disrupts Neural Progenitor Development and Leads to Microcephaly in Mice. (2016, background): “A causal association between ZIKV infection andmicrocephaly was proposed based on an increased incidence of microcephaly coinciding with the ZIKV outbreak and the detection of ZIKV in both microcephalic fetal brain tissues and the amniotic fluid of pregnant women with microcephalic fetuses (Calvet e”
- Zika Virus and Birth Defects--Reviewing the Evidence for Causality. (2016, positive): “Temporality Zika virus infection in mothers during pregnancy precedes the finding of microcephaly or other brain anomalies in fetuses or infants.(14-20)”
- Brain organoids: advances, applications and challenges (2019, background): “Despite the difficulty in collecting human brain samples with consistent quality, analyses of these samples have continued to expand our knowledge of normal and diseased human brains (Calvet et al., 2016; Hansen et al., 2010; Moore et al., 2017; Nowakowski et al., 2016; Ozair et al., 2018; Pollen et”
- A Mouse Model of Zika Virus Pathogenesis (2016, background): “This evidence includes detection of ZIKV RNA, full-length viral genome, or viral particles in the amniotic fluid or brains of fetuses diagnosed with microcephaly, as well as ZIKV IgM in amniotic fluid, consistent with fetal infection (Calvet et al., 2016; Martines et al., 2016; Mlakar et al., 2016; ”
- Brain Region-specific Organoids using Mini-bioreactors for Modeling ZIKV Exposure (2016, neutral): “Recent clinical studies have established that ZIKV can pass through placenta to gain access to the developing fetal brain (Calvet et al., 2016; Driggers et al., 2016; Mlakar et al., 2016).”
- Zika virus: History, emergence, biology, and prospects for control. (2016, background): “there was a higher incidence of MC in newborns from regions where a previous outbreak of ZIKVwas described; 2) virus has been detected in the amniotic fluid and tissues of >10 fetuses and newborns with microcephaly (Calvet et al., 2016; Mlakar et al., 2016) (Martines et al.”
- Zika Virus Depletes Neural Progenitors in Human Cerebral Organoids through Activation of the Innate Immune Receptor TLR3 (2016, neutral): “In fact, ZIKV was detected by electron microscopy and RT-qPCR in brains and amniotic fluid of microcephalic fetuses, strengthening the causal link between ZIKV and increased incidence of microcephaly (Calvet et al., 2016; Mlakar et al., 2016).”
- Zika Virus Infects Human Cortical Neural Precursors and Attenuates Their Growth (2016, positive): “ZIKVwas detected in the amniotic fluid of two pregnant women whose fetuses had been diagnosedwithmicrocephaly (Calvet et al., 2016), suggesting that ZIKV can cross the placental barrier.”
- Rapid, Low-Cost Detection of Zika Virus Using Programmable Biomolecular Components. (2016, positive): “Correlations between the increase in Zika virus infections, the development of fetal microcephaly (Calvet et al., 2016; Ga- lindo-Fraga et al., 2015; Victora et al., 2016), and Guillain-Barré syndrome have resulted in the declaration of a public health emergency by the World Health Organization…”