Increased hypothalamic GPR54 signaling: a potential mechanism for initiation of puberty in primates.
By M. Shahab, C. Mastronardi, S. Seminara, W. Crowley, S. Ojeda, T. Plant. Proceedings of the National Academy of Sciences of the United States of America, 2005
859 citations recorded by the source corpus.
Subfields: Endocrine and Autonomic Systems, Reproductive Medicine
View Paper: https://www.semanticscholar.org/paper/8574a024f2c5d4f84a52fe805c2a0e053abac0c3
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In Reproductive Medicine
Citations in the corpus
- Pubertal hormones organize the adolescent brain and behavior. (2005, background): “Most recently, kisspeptin and its cognate receptor GPR54 have been recognized as important players in the onset of puberty [54,75,107].”
- Kisspeptins and reproduction: physiological roles and regulatory mechanisms. (2012, background): “Similarly, hypothalamic Kiss1 mRNA levels raised in male monkeys along puberty (404).”
- Postnatal development of kisspeptin neurons in mouse hypothalamus; sexual dimorphism and projections to gonadotropin-releasing hormone (GnRH) neurons (2006, background): “Alongside the similarly widespread distribution of GPR54 mRNA in the hypothalamus (9), this observation indicates that kisspeptin-GPR54 signaling is very likely to be involved in the regulation of multiple hypothalamic networks.”