Aging, Progenitor Cell Exhaustion, and Atherosclerosis

By F. Rauscher, P. Goldschmidt-Clermont, B. Davis, Tao Wang, David Gregg, P. Ramaswami, A. Pippen, B. Annex, Chunming Dong, Doris A Taylor. Circulation, 2003

716 citations recorded by the source corpus.

Subfields: Genetics, Immunology, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/845b34f096c24a65ec7e36e71c438badbb10f92e

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Molecular Biology

1. A. Stolzing
2. A. Scutt

Citations in the corpus

• Aging of mesenchymal stem cells. (2006, background): “Correspondingly, some studies claim that MSC transplants from aged donors are less effective (Rauscher et al., 2003), others disagree (Dressler et al.”
• Impaired progenitor cell activity in age-related endothelial dysfunction. (2005, background): “It is therefore conceivable that any impairment of this vasculogenic element to endothelial regeneration may account for the progression of endothelial dysfunction and atherosclerosis ( 13 ).”
• Reduced Number of Circulating Endothelial Progenitor Cells Predicts Future Cardiovascular Events: Proof of Concept for the Clinical Importance of Endogenous Vascular Repair (2005, neutral): “Smoking 11 (26) 12 (27) 18 (54) ACS vs Co and CAD, 0.”
• Mechanisms of Vascular Aging (2018, background): “In particular, treatment with bone marrow-derived progenitor cells isolated from young mice prevents atherosclerosis progression in ApoE mice, whereas treatment with progenitor cells from older mice is ineffective.(234) It should be noted that role of EPCs in the genesis of vascular aging phenotypes”