Mediators of innate immune recognition of bacteria concentrate in lipid rafts and facilitate lipopolysaccharide-induced cell activation.

By M. Triantafilou, K. Miyake, D. Golenbock, K. Triantafilou. Journal of Cell Science, 2002

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Co-operation of TLR4 and raft proteins in LPS-induced pro-inflammatory signaling (2014, background): “Thus far, in support of the biochemical data, measurements of the fluorescence resonance energy transfer (FRET) between the constitutive raft components GM1 or CD14 and selected plasma membrane proteins have indicated that LPS induces the assembly of a raftassociated multimolecular complex composed ”
The grateful dead: damage‐associated molecular pattern molecules and reduction/oxidation regulate immunity (2007, background): “The importance of this process is demonstrated by the attenuation of LPS-dependent TLR4 activation upon disruption of the raft complex (80).”