MicroRNAs 103 and 107 regulate insulin sensitivity
By M. Trajkovski, Jean Hausser, J. Soutschek, B. Bhat, Akinc Akin, Mihaela Zavolan, M. Heim, M. Stoffel. Nature, 2011
1,006 citations recorded by the source corpus.
Subfields: Cancer Research, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/175c6a91c3c44368527bf50ced43b553845f9572
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Citations in the corpus
- Diabetic cardiomyopathy: an update of mechanisms contributing to this clinical entity (2018, background): “miR103, 107, -143, and -181 play a role in insulin sensitivity and systemic glucose metabolism.(145,146) Increased expression of miR-454, 500,-142-3p/5p, and 1246 has been identified in cardiac diastolic dysfunction.”
- Molecular mechanisms of diabetic cardiomyopathy (2014, neutral): “Recent reports suggested a role for miR-143, miR-181, miR-103, miR-107 and miR-802 in the regulation of systemic glucose metabolism and insulin sensitivity, thereby implicating miRNAs in the pathogenesis of insulin resistance and type 2 diabetes [124–126].”
- MicroRNAs in Common Human Diseases (2012, background): “Immune-related diseases Multiple sclerosis miR-145, miR-34a, miR-155, miR-326 [80,81] Systemic lupus erythematosus miR-146a [82,83] Type II diabetes miR-144, miR-146a, miR-150, miR-182, miR-103, miR-107 [84] Nonalcoholic fatty liver disease miR-200a, miR-200b, miR-429, miR-122, miR-451, miR-27 [86] ”