Caspases: pharmacological manipulation of cell death.

By Inna N. Lavrik, Alexander Golks, P. Krammer. Journal of Clinical Investigation, 2005

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How researchers see this paper

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Acrolein: sources, metabolism, and biomolecular interactions relevant to human health and disease. (2008, background): “Caspase-9 then activates the effector caspase-3 resulting in the execution of the cell-death program [129].”
Apoptosis in cancer: from pathogenesis to treatment (2011, background): “This is followed by a characteristic breakdown of DNA into large 50 to 300 kilobase pieces [18].”
Malignant astrocytic glioma: genetics, biology, and paths to treatment. (2007, background): “The “death receptors” are cell surface molecules that, upon binding their cognate ligands, recruit adapter molecules to provide a molecular scaffold for the autopro-teolytic processing and activation of caspases (for review, see Lavrik et al. 2005).”
Life and death in peripheral T cells (2007, background): “Further pro-apoptotic signals are delivered by proteolytic enzymes called caspases”