Gonadal steroid hormones: major regulators of gonadal sex differentiation and gametogenesis in fish

By Y. Nagahama. 2000

111 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/82747d0965c6d82de04d785d5b5368daa466464d

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Citations in the corpus

Morphology, endocrinology, and environmental modulation of gonadal sex differentiation in teleost fishes (2004, methodology): “Further evidence of steroid biosynthesis during sex differentiation was provided by immunohistochemical analysis of the gonads using antibodies developed against some of the enzymes (cholesterol side-chain cleavage cytochrome P450, 3β-hydroxysteroid dehydrogenase, cytochrome P450 17α-hydroxylase/17,”
Biomarkers of hypoxia exposure and reproductive function in Atlantic croaker: a review with some preliminary findings from the northern Gulf of Mexico hypoxic zone. (2009, background): “The side chain of cholesterol is cleaved by a P450 enzyme (P450scc) to produce a 21 carbon (C-21) steroid, pregnenolone, which is converted to C-21 steroid hormones, glucocorticoids and the two principal progestin hormones produced in fish, 17, 20β-dihydroxy-4-pregnen-3one (17, 20β-P) and 17, 20β,21”
More male fish embryos near a pulp mill (2000, background): “Thus, aromatase, which is the enzyme that converts androgen to estrogen, is thought to be involved in this process [13].”
Endocrine sex control strategies for the feminization of teleost fish (2001, background): “However, the existence of a single gene responsible for the development of one sex, like Sry is responsible for male Ž . differentiation in mammals, has not yet been found in fish Nagahama, 2000 .”
Sex determination and sex differentiation in fish: an overview of genetic, physiological, and environmental influences (2002, background): “The mechanism by which 17a, 20h DP causes egg maturation involves a complex interaction between oocyte 17a, 20h-DP receptors, prostaglandins PGE1, PGE2, PGF1a, PGF2a (Kobayashi and Stacey, 1993), and signal transduction pathways operating via inhibitory G proteins (Yoshikuni and Nagahama, 1994; Goet”