Helicobacter exploits integrin for type IV secretion and kinase activation

By T. Kwok, Dana Zabler, S. Urman, M. Rohde, R. Hartig, S. Wessler, R. Misselwitz, J. Berger, N. Sewald, W. König and 1 more. Nature, 2007

623 citations recorded by the source corpus.

Subfields: Immunology, Surgery

View Paper: https://www.semanticscholar.org/paper/7eeb3a454b7c99d536e5434035930d5fbd8da6c4

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In Immunology

1. R. Peek
2. Qiang Tian

In Surgery

1. R. Peek

Citations in the corpus

• Mechanisms of disease: Helicobacter pylori virulence factors (2010, neutral): “T4SS-injected CagA primarily localizes to focal adhesions”
• Human Gut Microbiota and Gastrointestinal Cancer (2018, background): “Cag strain infections present a highly increased risk of gastric cancers [36,37].”
• Helicobacter pylori: gastric cancer and beyond (2010, background): “Integrin receptors on host cells represent a portalof entry for CagA injectio”
• Biological Diversity of Prokaryotic Type IV Secretion Systems (2009, background): “pylori Cag system, a number of Mpf subunits designated “VirB-like” in fact bear little resemblance to the VirBAt proteins (17, 164, 227, 256) (Fig.”