Molecular mechanisms of calcium-dependent neurodegeneration in excitotoxicity.
By M. Arundine, M. Tymianski. Cell Calcium, 2003
841 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Molecular Biology, Pharmacology
View Paper: https://www.semanticscholar.org/paper/7ecacc5d5f85fba555e9a4247b90ad2bfaaafd3a
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Cellular and Molecular Neuroscience
- Mark P. Mattson
- Beth Levine
- Guido Kroemer
- Valina L. Dawson
- T. Dawson
- J. Martinou
- L. Galluzzi
- R. Rizzuto
In Molecular Biology
- Guido Kroemer
- Douglas R. Green
- L. Galluzzi
- Beth Levine
- Simone Fulda
- E. White
- N. Mizushima
- Peter Vandenabeele
In Pharmacology
- Guido Kroemer
- Beth Levine
- L. Galluzzi
- Douglas R. Green
- N. Mizushima
- E. White
- Eric H. Baehrecke
- J. M. Bravo-San Pedro
Citations in the corpus
- Synaptic and extrasynaptic NMDA receptors are gated by different endogenous coagonists. (2012, background): “They are also central to the path- ogenesis of various neurological and psychiatric diseases, including neurodegenerative disorders (Arundine and Tymianski, 2003), chronic pain (Woolf and Salter, 2000), and schizophrenia (Lin et al., 2012).”
- Essential versus accessory aspects of cell death: recommendations of the NCCD 2015 (2014, background): “Besides inhibiting system xC , glutamate can trigger MPT-driven RCD upon the hyperactivation of ionotropic receptors, a neurotoxic process commonly known as excitotoxicity.(250,251) Cell death: causes versus correlates L Galluzzi et al”
- Calcium and neurodegeneration (2007, background): “Thus, oxidative stress renders neurons vulnerable to a form of Ca 2+ - mediated death called excitotoxicity in which glutamate receptors are overactivated even as the intracellular Ca 2+ concentrations rise beyond tolerable levels (Arundine & Tymianski, 2003).”