Glucosinolate metabolism and its control.
By C. Grubb, S. Abel. Trends in Plant Science, 2006
705 citations recorded by the source corpus.
Subfields: Molecular Biology, Plant Science
View Paper: https://www.semanticscholar.org/paper/7e87e908555886d2a85c79340413fadc83052977
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Citations in the corpus
- Secondary Metabolites of Plants and their Role: Overview (2015, background): “(III) Sulphur containing secondary metabolites: They include GSH, GSL, Phytoalexins, Thionins, defensins and allinin which have been linked directly or indirectly with the defence of plants against microbial pathogens (12,13,14).”
- Glucosinolate Biosynthesis and the Glucosinolate–Myrosinase System in Plant Defense (2020, background): “CYP83A1 converts the aldoximes to unidentified intermediates (previously assumed as aci-nitro compounds) [36], which are conjugated with glutathione (GSH) by glutathione S-transferase F11 (GSTF11) and GSTU20, and then cleaved by gamma-glutamyl peptidase 1 (GGP1) and C-S lyase (Super root 1 (SUR1)) t”
- The ‘mustard oil bomb’: not so easy to assemble?! Localization, expression and distribution of the components of the myrosinase enzyme system (2008, background): “The biosynthesis and transport of glucosinolates as well as the regulation of glucosinolate metabolism will not be discussed here and we refer the reader to recent reviews covering these aspects (Grubb and Abel 2006; Halkier and Gershenzon 2006; Yan and Chen 2007).”
- Role of secondary metabolites in defense mechanisms of plants (2011, background): “…aglycon rearranges with loss of the sulphate to give pungent and chemically reactive products, including isothiocyanates and nitriles, function in defense as herbivorous toxins and feeding repellent (Renwick, 1992; Grubb and Abel, 2006; Halkier and Gershenzon, 2006; Talalay and Fahey, 2001).”
- Glucosinolate profiling of seeds and sprouts of B. oleracea varieties used for food (2007, background): “Glucosinolates are plant defence compounds and consistent with this function are accumulated preferentially in the organs that contribute most to plant fitness at a particular moment in the growth cycle (Brown et al., 2003; Grubb and Abel, 2006; Halkier and Gershenzon, 2006).”
- Metabolism and Plant Hormone Action During Clubroot Disease (2009, background): “The general pathways for GSL biosynthesis have been described elsewhere (Grubb and Abel 2006; Halkier and Gershenzon 2006).”
- Root and shoot glucosinolates: a comparison of their diversity, function and interactions in natural and managed ecosystems (2008, background): “For example, there is a tight direct link between indole GSL biosynthesis and the principal auxin, indol-3-acetic acid (IAA) metabolism; they are both derived from tryptophan and they share the first dedicated step in their biosynthesis (reviewed in Grubb and Abel 2006 ).”
- Phytochemistry and biological activity of mustard (Brassica juncea): a review (2020, neutral): “The biosynthesis of glucosinolates consists of three components: elongation of the amino acid side chain, formation of the core structure, and modification of the secondary side chain (Grubb & Abel, 2006).”
- Specific and coordinated control of indolic and aliphatic glucosinolate biosynthesis by R2R3-MYB transcription factors in Arabidopsis thaliana (2008, background): “Glucosinolates encompass the amino acid-derived class of sulfur and nitrogen containing secondary metabolites, characteristic of the order Brassicales, which include brassica vegetables, oilseed rape and the model plant Arabidopsis thaliana ( Grubb and Abel 2006; Halkier and Gershenzon 2006).”
- MYC2 Differentially Modulates Diverse Jasmonate-Dependent Functions in Arabidopsis[W] (2007, methodology): “Intact IGs and glucosinolates, in general, are thought to be nontoxic, but their breakdown products, isothiocyanates and nitriles, can be toxic (reviewed in Wittstock and Halkier, 2002; Grubb and Abel, 2006).”
- Metabolite profiling of symbiont and host during thermal stress and bleaching in the coral Acropora aspera (2017, background): “Glucosinolates are formed via a portion of the shikimate pathway, which is present in both partners (Grubb and Abel 2006; Rosic and Dove 2011).”