Risk of hepatocellular carcinoma across a biological gradient of serum hepatitis B virus DNA level.

By Chien-Jen Chen, Hwai I. Yang, Jun Su, C. Jen, S. You, Sheng-Nan Lu, Guan-Tarn Huang, U. Iloeje. Journal of the American Medical Association (JAMA), 2006

3,131 citations recorded by the source corpus.

Subfields: Epidemiology, Hepatology

View Paper: https://www.semanticscholar.org/paper/7be5321715632d1c479416ecc6831590b19bc325

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In Hepatology

Citations in the corpus

Diagnosis, Staging, and Management of Hepatocellular Carcinoma: 2018 Practice Guidance by the American Association for the Study of Liver Diseases (2018, background): “(25) Active viral replication is associated with higher risk of HCC, and long-standing active infection with inflammation resulting in cirrhosis is the major event resulting in increased risk.(26,27) The incidence of HCC in inactive HBV carriers without liver cirrhosis (LC) is less than 0.”
Hepatocellular carcinoma pathogenesis: from genes to environment (2006, background): “The impact of HBV infection on HCC development is reflected by the correlation between increased incidence of HCC in patients with increasing levels of HBV DNA in seru”
Diagnosis, Staging, and Management of Hepatocellular Carcinoma: 2018 Practice Guidance by the American Association for the Study of Liver Diseases (2019, background): “(25) Active viral replication is associated with higher risk of HCC, and long-standing active infection with inflammation resulting in cirrhosis is the major event resulting in increased risk.(26,27) The incidence of HCC in inactive HBV carriers without liver cirrhosis (LC) is less than 0.”