Effect of left ventricular outflow tract obstruction on clinical outcome in hypertrophic cardiomyopathy.

By M. Maron, Iacopo Olivotto, S. Betocchi, S. Casey, John R. Lesser, M. Losi, F. Cecchi, B. Maron. New England Journal of Medicine, 2003

1,388 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Radiology, Nuclear Medicine and Imaging

View Paper: https://www.semanticscholar.org/paper/7a8eb4763e062ca1ed433f5c1041543456fed00e

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cardiology and Cardiovascular Medicine

1. B. Maron
2. Steve R. Ommen
3. M. Maron
4. P. Spirito
5. C. Semsarian
6. Iacopo Olivotto
7. Mark S. Link
8. C. Seidman

In Radiology, Nuclear Medicine and Imaging

1. Detlef Lohse
2. Xuehua Zhang

Citations in the corpus

• Abnormal Calcium Handling Properties Underlie Familial Hypertrophic Cardiomyopathy Pathology in Patient-Specific Induced Pluripotent Stem Cells (2013, background): “Patients with HCM exhibit abnormal thickening of the left ventricular (LV) myocardium in the absence of increased hemodynamic burden and are at heightened risk for clinical complications such as progressive heart failure, arrhythmia, and sudden cardiac death (SCD) (Maron, 2002; Maron et al., 2003).”
• Hypertrophic Cardiomyopathy Is Predominantly a Disease of Left Ventricular Outflow Tract Obstruction (2006, neutral): “LVOT gradient with Valsalva 30 mm Hg, n (%) 0 4 (13) 13 (17) 0.”
• A novel clinical risk prediction model for sudden cardiac death in hypertrophic cardiomyopathy (HCM risk-SCD). (2014, background): “The pre-specified predictors, their definitions, and coding are summarized in Table 1.(15,17,18,20,21,23) –31 Even though both left atrial (LA) size and atrial fibrillation (AF) are associated with SCD,(31,32) only the former was included as a pre-specified predictor since LA enlargement predisposes”
• Hypertrophic cardiomyopathy: present and future, with translation into contemporary cardiovascular medicine. (2014, background): “Outflow gradients are characteristically dynamic, with spontaneous variability influenced by altered myocardial contractility and loading conditions (2,4,11,16,21,39,40).”
• American College of Cardiology/European Society of Cardiology clinical expert consensus document on hypertrophic cardiomyopathy. A report of the American College of Cardiology Foundation Task Force on Clinical Expert Consensus Documents and the European Society of Cardiology Committee for Practice G (2003, background): “Although it has previously been subject to periodic controversy (72,120,125,126), there is now widespread recognition that the subaortic gradient (30 mm Hg or more) and associated elevations in intra-cavity LV pressure reflect true mechanical impedance to outflow and are of pathophysiologic and prog”