Beta oscillatory activity in the subthalamic nucleus and its relation to dopaminergic response in Parkinson's disease.

By M. Weinberger, N. Mahant, W. Hutchison, Andres M. Lozano, E. Moro, M. Hodaie, A. Lang, J. Dostrovsky. Journal of Neurophysiology, 2006

595 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Neurology

View Paper: https://www.semanticscholar.org/paper/7a6370a51b19c1d63d4f5952ed0566309537167e

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cellular and Molecular Neuroscience

1. Morten L. Kringelbach
2. Peter Brown
3. Hagai Bergman
4. Ludvic U. Zrinzo
5. Ned Jenkinson
6. T. Foltynie
7. Kent C. Berridge
8. Ruth Feldman

In Neurology

1. Morten L. Kringelbach
2. Peter Brown
3. Ned Jenkinson
4. Hagai Bergman
5. Ludvic U. Zrinzo
6. T. Foltynie

Citations in the corpus

• Oscillations and the basal ganglia: Motor control and beyond (2013, unknown): “We have, for example, already touched upon the robust correlations that have been reported between treatment induced suppressions of beta LFP activity in the STN and improvements in bradykinesia and rigidity (Kühn et al., 2006, 2008, 2009; Ray et al., 2008; Weinberger et al., 2006).”
• Translational principles of deep brain stimulation (2007, background): “Further evidence of the origin of these oscillations was recently obtained in a study of intraoperative LFP and neuronal spike recordings in the STN of fourteen patients with Parkinson's disease, which showed that the LFP beta oscillatory activity is generated mostly through spiking activity within ”
• High-Frequency Stimulation of the Subthalamic Nucleus Suppresses Oscillatory β Activity in Patients with Parkinson's Disease in Parallel with Improvement in Motor Performance (2008, unknown): “…has been inferred from oscillations in local field potentials (LFPs), and several lines of evidence support the use of such activity as a surrogate measure of neuronal synchrony in the basal ganglia (Levy et al., 2002; Brown and Williams, 2005; Kühn et al., 2005; Weinberger et al., 2006).”
• Pathological synchronization in Parkinson's disease: networks, models and treatments. (2007, background): “In these clinical studies, it is conspicuous that treatment-induced suppression of synchrony does not correlate with the degree of improvement in rest tremor, and an early report of an association between beta band activity in the basal ganglia and tremor was not replicated in a larger study from th”
• Deep brain stimulation can suppress pathological synchronisation in parkinsonian patients (2010, background): “The present study suffers from several limitations. In order to recruit a large patient sample, we studied patients at several different surgical centres, which may have introduced additional variance in our data set. Another limitation was the absence of kinematic assessment that might have allowed”