Retinoid Signaling Determines Germ Cell Fate in Mice
By J. Bowles, D. Knight, Christopher Smith, D. Wilhelm, J. Richman, S. Mamiya, K. Yashiro, K. Chawengsaksophak, M. Wilson, Janet Rossant and 2 more. Science, 2006
1,013 citations recorded by the source corpus.
Subfields: Genetics, Molecular Biology, Reproductive Medicine
View Paper: https://www.semanticscholar.org/paper/78f24981c8cb84e1869f9f4ca3199983d122243c
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Citations in the corpus
- Oogenesis in teleosts: how eggs are formed. (2010, background): “bules that are connected to the gonad at the anterior end (Bowles et al., 2006; Bowles and Koopman, 2007).”
- Epigenetic events in mammalian germ-cell development: reprogramming and beyond (2008, background): “A number of environmental cues, including retinoic acid signals from the mesonephro”
- Spermatogenesis in fish. (2010, background): “In the foetal testis, however, after a brief period of proliferation the gonocytes are prevented from entering meiosis by the action of retinoid-degrading enzyme CYP26B1 expressed by Sertoli cells (Bowles et al., 2006).”
- The mammalian ovary from genesis to revelation. (2009, background): “5 but up-regulated in the testis, thus allowing accessibility of the XX germ cells, but not the XY germ cells, to retinoic acid (105).”
- Spermatogenesis: The Commitment to Meiosis. (2016, background): “Some meiosis-specific chromatin changes in germ cells have been reported to occur shortly after the formation of the gonad, and access to RA at this time results in premature entry of germ cells into meiosis (8, 71).”
- Fish Gonadogenesis. Part II: Molecular Biology and Genomics of Sex Differentiation (2008, background): “Further, the recent findings of Koubova et al. (2006) and Bowles et al.”