Rho GTPases and the control of cell behaviour.

By A. Hall. Biochemical Society Transactions, 2005

829 citations recorded by the source corpus.

Subfields: Cell Biology, Immunology and Allergy, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/7791219199d72a6be9d7160c36016f4551de9251

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cell Biology

1. Christopher S. Chen
2. C. Carraher
3. Purva Singh
4. Masanori Nakayama
5. M. Maeda
6. M. Amano
7. Daniel M. Cohen
8. M. Wheelock

In Immunology and Allergy

1. C. Carraher
2. Purva Singh
3. Christopher S. Chen
4. Michael T. Yang
5. J. Schwarzbauer
6. Gerhard Christofori
7. Mahmut Yilmaz
8. Wen-Sheng Wu

In Molecular Biology

1. E. Taverna
2. W. Huttner
3. M. Götz
4. Mahmut Yilmaz
5. Christopher S. Chen
6. Masanori Nakayama
7. M. Maeda
8. M. Amano

Citations in the corpus

• EMT, the cytoskeleton, and cancer cell invasion (2009, background): “With regard to migration and invasion and in a simplified view, RhoA induces actin stress fiber formation and regulates cytoskeletal changes affecting cell-cell or cell-matrix adhesion, Rac1 is involved in lamellipodia and membrane ruffle formation, and Cdc42 is involved in filopodia formation [136,”
• The signaling mechanism of ROS in tumor progression (2007, background): “small GTPase Rac, well known to be essential for cytoskeletal rearrangement [58], is the most important”
• Cadherin switching (2008, background): “Rho GTPases in cadherin-mediated phenotypic transformations The Rho family of small GTPases regulate cell motility, as well as many other cellular functions (Hall, 2005; Jaffe and Hall, 2005; Raftopoulou and Hall, 2004).”
• The cell biology of neurogenesis: toward an understanding of the development and evolution of the neocortex. (2014, background): “Finally, cdc42 is also involved in regulating various signaling pathways, such as integrin signaling or Par-complex signaling (Hall 2005, Jaffe & Hall 2005, Tepass 2012).”
• Assembly of Fibronectin Extracellular Matrix (2010, background): “Rho-GTP then stimulates Rho kinases to enhance cell contractility by inducing actin-myosin interactions and actin rearrangement into stress fibers (Hall 2005).”
• Mechanical tugging force regulates the size of cell–cell junctions (2010, background): “In either model, junction growth is predicated on membrane protrusive activity, a Rac-dependent process (47).”
• Rho-Kinase/ROCK: A Key Regulator of the Cytoskeleton and Cell Polarity (2010, background): “Rho regulates stress fiber formation and cell contraction, whereas Rac and Cdc42 regulate the formation of lamellipodia and filopodia, respectively, and promote protrusive activities [Hall, 2005].”