Noncytolytic control of viral infections by the innate and adaptive immune response.

By L. Guidotti, F. Chisari. Annual Review of Immunology, 2001

1,077 citations recorded by the source corpus.

View Paper: https://www.semanticscholar.org/paper/7585ffcbe4659c8e12b385c8f13d8c2c36ecc13c

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Citations in the corpus

Adenovirus Infections in Immunocompetent and Immunocompromised Patients (2014, background): “Moreover, innate effector cells, particularly natural killer cells, which can destroy virus-infected cells in a nonspecific fashion, are recruited and activated (104, 113).”
Recognition of double-stranded RNA and activation of NF-κB by Toll-like receptor 3 (2001, background): “Viral infection of mammalian cells results in activation of an innate immune response mediated by type I IFNs, IFN-α and IFN-β, and other cytokines, including interleukin (IL)-6 and IL-12 (refs&nbs”
Adoptive-cell-transfer therapy for the treatment of patients with cancer (2003, background): “cgi?l=8743">TRAIL )) and cytotoxic degranulation, which leads to perforinmediated lysi”
Immunobiology and pathogenesis of viral hepatitis. (2006, background): “Like innate immune cells described above, these T cells participate in viral pathogenesis either directly (by killing infected cells) or indirectly (by producing soluble factors such as cytokines and chemokines) that contribute to the inflammatory process and/or inhibit viral replication (1, 94).”
Occult hepatitis B virus infection. (2007, background): “In fact, there is evidence indicating that cytokines like tumor necrosis factor a and interferon c may strongly inhibit the HBV gene expression at post-transcriptional level [37,38].”
Viruses and interferon: a fight for supremacy (2002, background): “Both class-I-restricted CD8 + T cells and class-II-restricted CD4 + T cells are activated during viral infectio”