Glycogen synthase kinase-3β mediates convergence of protection signaling to inhibit the mitochondrial permeability transition pore
By M. Juhaszova, D. Zorov, Suhn Hee Kim, S. Pepe, Qin Fu, K. Fishbein, B. Ziman, Su-yang Wang, K. Ytrehus, C. Antos and 2 more. 2004
925 citations recorded by the source corpus.
Subfields: Molecular Biology, Pathology and Forensic Medicine
View Paper: https://www.semanticscholar.org/paper/747f7b2c2c8c0594139d5f7eb2ee5143b49967a6
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In Molecular Biology
In Pathology and Forensic Medicine
Citations in the corpus
- Chronic inhibition of cyclic GMP phosphodiesterase 5A prevents and reverses cardiac hypertrophy (2005, background): “, which is phosphorylated by Akt as well as other kinase”
- Interaction of Cardiovascular Risk Factors with Myocardial Ischemia/Reperfusion Injury, Preconditioning, and Postconditioning (2007, background): “More recent interest has focused on glycogen synthase-3 (GSK-3 ) as a distal kinase, phosphorylated (and hence inactivated) by other kinases including Akt and p42/p44 MAPK/ERK (Tong et al., 2002; Juhaszova et al., 2004).”