Acute kidney injury increases risk of ESRD among elderly.

By A. Ishani, J. Xue, Jonathan Himmelfarb, Paul Eggers, P. Kimmel, B. Molitoris, A. Collins. Journal of the American Society of Nephrology, 2009

1,131 citations recorded by the source corpus.

Subfields: Nephrology

View Paper: https://www.semanticscholar.org/paper/746033d2e0abecba8a5871de1de823b6e51e440f

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In Nephrology

1. L. Chawla
2. P. Kimmel
3. Joseph V. Bonventre
4. Jeremy S. Duffield

Citations in the corpus

• Epithelial cell cycle arrest in G2/M mediates kidney fibrosis after injury (2010, positive): “Acute kidney injury has been recognized as a major contributor to chronic and end-stage kidney diseas”
• Acute kidney injury and chronic kidney disease: an integrated clinical syndrome. (2012, methodology): “James et al.23 showed that patients undergoing percutaneous coronary revascularization who had AKI were at risk for future development of ESRD.”
• KDOQI US commentary on the 2012 KDIGO clinical practice guideline for acute kidney injury. (2013, background): “650 on CKD is associated with acceleration in the rate of progression to end-stage disease.(13-17) Our understanding of the epidemiology of AKI and interpretation of results across clinical trials has been hindered by the prior absence of a broadly accepted clinical definition, with more than 30 ope”
• Pathophysiology of Acute Kidney Injury (2012, neutral): “develop AKI and to have a less successful outcome (93, 227, 479, 480).”
• Chronic dialysis and death among survivors of acute kidney injury requiring dialysis. (2009, methodology): “Ishani et al previously used American Medicare administrative data to identify approximately 7000 patients older than 67 years with hospital-associated acute kidney injury who survived to discharge.(22) Postdischarge mortality was doubled and the risk of chronic dialysis was increased by nearly 7 ti”
• THE AGING KIDNEY: PHYSIOLOGICAL CHANGES (2010, background): “While the systemic renin-angiotensin system (RAS) is suppressed in aging, the intrarenal RAS may not be equivalently suppressed (45,46), and pharmacologic RAS blockade has been shown to slow the progression of age-related CKD (19,47).”