Cutting Edge: Differential Roles for Phosphoinositide 3-Kinases, p110γ and p110δ, in Lymphocyte Chemotaxis and Homing1

By K. Reif, K. Okkenhaug, Takehiko Sasaki, Josef M. Penninger, B. Vanhaesebroeck, J. Cyster. Journal of Immunology, 2004

229 citations recorded by the source corpus.

Subfields: Immunology, Molecular Biology, Oncology

View Paper: https://www.semanticscholar.org/paper/73fc4a0ab8ff1358b8b3e697cf7d53ea3b8ef71c

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Immunology

1. J. Cyster
2. Christopher D. C. Allen
3. R. Lesley
4. K. Ansel
5. N. Killeen
6. N. Haynes
7. Shane Crotty
8. A. Luster

In Molecular Biology

1. K. Okkenhaug
2. J. Cyster
3. B. Vanhaesebroeck
4. A. Luster

In Oncology

1. Christopher D. C. Allen
2. J. Cyster

Citations in the corpus

• The p110δ Isoform of Phosphoinositide 3-Kinase Controls Clonal Expansion and Differentiation of Th Cells1 (2006, background): “4A, day 0 time point), consistent with a nonessential role for p110 in T cell migration (31).”
• Role of CXCR5 and CCR7 in Follicular Th Cell Positioning and Appearance of a Programmed Cell Death Gene-1High Germinal Center-Associated Subpopulation1 (2007, background): “The finding that T and B cells differ in their PI3K and DOCK2 use when responding to lymphoid chemokines provides a possible explanation for the differences in CXCL13 chemokine sensitivity of the two cell types (45, 46).”