The peripheral benzodiazepine binding site in the brain in multiple sclerosis: quantitative in vivo imaging of microglia as a measure of disease activity.

By R. Banati, J. Newcombe, Roger N. Gunn, A. Cagnin, Federico E. Turkheimer, Frank Heppner, G. Price, F. Wegner, G. Giovannoni, D. Miller and 8 more. Brain : a journal of neurology, 2000

683 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Neurology, Pathology and Forensic Medicine

View Paper: https://www.semanticscholar.org/paper/737ff5bd8bf6771cb355e586547443ac76c487f3

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cellular and Molecular Neuroscience

1. M. Block
2. Jau-Shyong Hong
3. Paul M. Matthews

In Neurology

1. M. Block
2. Jau-Shyong Hong
3. Manuel B. Graeber
4. Roger N. Gunn
5. Eugenii A. Rabiner
6. P. Whitton
7. Melinda E Lull

In Pathology and Forensic Medicine

1. Roger N. Gunn
2. Eugenii A. Rabiner

Citations in the corpus

• An 18-kDa Translocator Protein (TSPO) polymorphism explains differences in binding affinity of the PET radioligand PBR28 (2011, background): “The poor signal-to-noise ratio and high nonspecific binding of the first-generation ligand [11C]PK11195 limit accurate quantification (Banati et al, 2000).”
• Microglia and inflammation-mediated neurodegeneration: multiple triggers with a common mechanism. (2005, methodology): “Using nuclear magnetic resonance imaging, positron emission tomography, and [11C](R)-PK11195 (a microglia marker) Banati et al. (2000) analyzed the brain of MS patients and showed increased microglia activity around the site of the MS lesion (Banati et al., 2000).”
• Microglia: biology and pathology (2009, background): “possible to study microglia activation in vivo [6, 7, 24].”