Global Epigenomic Reconfiguration During Mammalian Brain Development

By Ryan Lister, Eran A. Mukamel, Joseph R. Nery, M. Urich, C. Puddifoot, Nicholas D. Johnson, J. Lucero, Yun Huang, A. Dwork, Matthew D. Schultz and 12 more. Science, 2013

1,784 citations recorded by the source corpus.

Subfields: Genetics, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/70874f98fbd92606e00f31cfa61783af0b141def

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Citations in the corpus

EDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting Chemicals. (2015, background): “DNA methylation is a process in which methyl groups are attached to cytosine residues by DNA methyltransferase enzymes (DNMTs), usually in cytosine-guanosine dinucleotide pairs (CpG sites), although DNA methylation can occur on non-CpG residues (82, 83).”
Editing DNA methylation in the mammalian genome (2016, neutral): “Owing to the advancement in sequencing technologies, single-nucleotide resolution methylation maps for many types of human and mouse cells and tissues have been depicted (Lister et al., 2009, 2013).”
Landscape of X chromosome inactivation across human tissues (2016, positive): “Others have used bias in DNA methylation 3,4,9 or in gene expression 5,10 between males and females as a proxy for XCI status.”
Physiology of Astroglia. (2018, neutral): “Moreover, epigenetic regulation of astroglia seems to be similarly different: the glial methylome remains more fetal and hence labile during adulthood (989).”
DNA methylation and healthy human aging (2015, background): “Therefore, tissue-specific epigenetic clock sites in some of these studies may not be accurate; however, the overall patterns of DNA methylation change with age have been validated in studies that did correct for cellular composition (Lam et al., 2012; Lister et al., 2013; Jaffe & Irizarry, 2014).”
Reversing DNA Methylation: Mechanisms, Genomics, and Biological Functions (2014, neutral): “Interestingly, base-resolution mapping indicates that 5hmC is actually depleted at the binding motif of transcription factors or CTCF insulators but enriched at immediately adjacent regions (Lister et al., 2013; Yu et al., 2012).”