Receptors, Mediators, and Mechanisms Involved in Bacterial Sepsis and Septic Shock

By E. V. van Amersfoort, T. V. van Berkel, J. Kuiper. Clinical Microbiology Reviews, 2003

782 citations recorded by the source corpus.

Subfields: Immunology, Microbiology, Public Health, Environmental and Occupational Health

View Paper: https://www.semanticscholar.org/paper/70793fd00373bc18fb8065c06e8f78993336cfe4

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In Immunology

1. S. Kanoh
2. B. Rubin

Citations in the corpus

• IL-6 mediates hypoferremia of inflammation by inducing the synthesis of the iron regulatory hormone hepcidin. (2004, neutral): “( B ) Hep3B cells: untreated (0), treated for 24 hours with 16.6% medium from monocyte-derived macrophages ( n = 2 donors) or Kupffer cells ( n = 2 donors) either unstimulated (M) or stimulated with 100 ng/ml LPS (M-LPS) or treated for 24 hours with the same 16.6% M-LPS + 6 μ g/ml anti–IL-6 Ab's (M-”
• Mechanisms of Action and Clinical Application of Macrolides as Immunomodulatory Medications (2010, background): “The parenchyma injury is associated with overexpression of cytokines and inflammatory mediators and with upregulation of adhesion molecules in vascular beds (40, 310, 320).”