Copper-zinc superoxide dismutase and amyotrophic lateral sclerosis.

By J. Valentine, P. A. Doucette, Soshanna Zittin Potter. Annual Review of Biochemistry, 2005

710 citations recorded by the source corpus.

Subfields: Genetics, Neurology

View Paper: https://www.semanticscholar.org/paper/6fbdb938f8c65a73d5c17b52247dd8ef205e4211

How researchers see this paper

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In Genetics

1. Robert H. Brown
2. P. Pasinelli

In Neurology

1. Robert H. Brown
2. P. Pasinelli

Citations in the corpus

• Wild-type and mutant SOD1 share an aberrant conformation and a common pathogenic pathway in ALS (2010, background): “In FALS, cytotoxicity of motor neurons appears to result from a gain of toxic SOD1 function, rather than from a loss of dismutase activit”
• Regulation of superoxide dismutase genes: implications in disease. (2009, background): “sod1-knockout mice have been shown to suffer from physiological impairments including reduced female fertility, macular degeneration, and death from liver tumors [131,136].”