Liver inflammation and fibrosis.
By Yukinori Koyama, D. Brenner. Journal of Clinical Investigation, 2017
1,044 citations recorded by the source corpus.
Subfields: Epidemiology, Hepatology, Surgery
View Paper: https://www.semanticscholar.org/paper/6eb5254e4b39fa07c215061b664c342814ef582a
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Epidemiology
- S. Friedman
- Jerrold M. Olefsky
- J. Iredale
- Gerald I. Shulman
- Alan R. Saltiel
- J. Pollard
- R. Loomba
- TA Wynn
In Hepatology
In Surgery
Citations in the corpus
- Resolving the fibrotic niche of human liver cirrhosis at single cell level (2019, background): “331 Disease-specific endothelial cells Endo(6) and Endo(7), CD34+PLVAP+VWA1+ and 332 CD34+PLVAP+ACKR1+ respectively (Fig.”
- Mechanisms and disease consequences of nonalcoholic fatty liver disease (2021, background): “Recent articles have reviewed general pathways of fibrosis and its degradation in the liver (Koyama and Brenner, 2017; Tsuchida and Friedman, 2017), and here we focus primarily on NASH-specific mechanisms.”
- Inflammatory mechanisms linking obesity and metabolic disease (2017, background): “…discuss the close connection between obesity, diabetes, hepatic inflammation, and the development of nonalcoholic fatty liver disease (NAFLD), which can progress to nonalcoholic steatohepatitis (NASH) and eventually frank cirrhosis due to sequential activation of inflammation and fibrosis (10).”