Studies of clomazone mode of action

By Y. Ferhatoglu, M. Barrett. 2006

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Biochemical Markers and Enzyme Assays for Herbicide Mode of Action and Resistance Studies (2015, background): “The oxidation of clomazone to ketoclomazone is catalyzed by a P450 monooxygenase, and treatment of plants with phorate or other P450 monooxygenase inhibitors reduces the metabolism of clomazone and protects plants from bleaching (Ferhatoglu and Barrett 2006; Ferhatoglu et al. 2005).”
Chlorophyll fluorescence as a marker for herbicide mechanisms of action (2012, background): “This herbicide targets 1-deoxy-D-xylulose-5-phosphate synthase (DXS), a key enzyme in the methyl-erythritol phosphate (MEP) isoprenoid pathway [36–38].”
Plant cell membrane as a marker for light-dependent and light-independent herbicide mechanisms of action. (2011, background): “This metabolite is a potent inhibitor of the first step in the plastid-specific, MEP terpenoid pathway, 1-deoxy-D-xylulose-5-phosphate synthase [30,31].”
Herbicide Metabolism: Crop Selectivity, Bioactivation, Weed Resistance, and Regulation (2019, background): “OH clomazone, inhibits plant 1-deoxy-D-xylulose-5-phosphate synthase (DXP synthase; Ferhatoglu and Barrett 2006), the first step in this pathway.”