Exploration of tumor-suppressive microRNAs silenced by DNA hypermethylation in oral cancer.
By K. Kozaki, I. Imoto, S. Mogi, K. Omura, J. Inazawa. Cancer Research, 2008
633 citations recorded by the source corpus.
Subfields: Cancer Research, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/6cebf79bec2c4aa6a0b58f5cf0547f5abef1f45c
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In Cancer Research
In Molecular Biology
Citations in the corpus
- Inactivation of miR-34a by aberrant CpG methylation in multiple types of cancer (2008, background): “In three PCa specimens (12, 17, 23) high expression of miR-34a in carcinoma cells was accompanied by the absence of CpG methylation in miR-34a.”
- Aberrant epigenetic landscape in cancer: how cellular identity goes awry. (2010, methodology): “Indeed, several groups have now used strategies based on the treatment of cancer cell lineswithDNA-demethylating reagents and/or histone deacetylase inhibitors in vitro to study changes inmiRNAexpression (Lujambio et al., 2008;Grady et al., 2008; Kozaki et al., 2008; Furuta et al., 2010).”