Plant responses to abiotic stresses: heavy metal-induced oxidative stress and protection by mycorrhization.

By A. Schützendübel, A. Polle. Journal of Experimental Botany, 2002

2,214 citations recorded by the source corpus.

Subfields: Analytical Chemistry, Plant Science, Pollution

View Paper: https://www.semanticscholar.org/paper/67c7bb06830122b27a7977a5f43ebfd2f6517629

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In Analytical Chemistry

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Citations in the corpus

Plant Responses to Heavy Metal Toxicity (2012, background): “If the intracellular concentration of metal ions saturates the defense mechanisms discussed above then the plant will begin to suffer oxidative stress caused by the production of ROS and the inhibition of metal-dependent antioxidant enzymes (Schützendübel and Polle 2002).”
Effect of 28-homobrassinolide treatment on nickel toxicity in Brassica juncea (2007, background): “The defence systems are (a) the metabolites including ascorbate, glutathione, tocopherol, and proline, and (b) enzymatic scavengers, include superoxide dismutases, CAT, PER, and glutathione reductases (Schützendübel and Polle 2002).”
Phenolics metabolism and lignin synthesis in root suspension cultures of Panax ginseng in response to copper stress (2006, background): “Cu may generate oxidative injury by inducing the production of reactive oxygen species (ROS) [1,2], which include superoxide anion (O2 ), singlet oxygen (O2) and hydrogen peroxide (H2O2).”
Abscisic acid accumulation and cadmium tolerance in rice seedlings (2005, background): “124, 2005 75 recent review, Schützendübel and Polle (2002) suggested that the depletion of GSH was a critical step in Cd-induced AOS generation.”
Antioxidant Enzyme Activities and Abiotic Stress Tolerance Relationship in Vegetable Crops (2016, positive): “It was recorded by Schützendübel and Polle [110] that antioxidative enzymes were prevented from functioning and that GSH was depleted for a short period by cadmium and other metals.”
Definition of “Heavy Metals” and Their Role in Biological Systems (2010, positive): “• It has been shown on several occasions that lipid peroxidation (Bertrand and Poirier 2005) is just a consequence of oxidative stress, such as that caused by glutathione depletion (Schützendübel and Polle 2002).”