The DRASIC cation channel contributes to the detection of cutaneous touch and acid stimuli in mice.
By M. Price, S. McIlwrath, Jinghui Xie, Chun Cheng, J. Qiao, Deirdre E. Tarr, K. Sluka, T. Brennan, G. Lewin, Michael J. Welsh. Neuron, 2001
614 citations recorded by the source corpus.
Subfields: Molecular Biology, Physiology, Sensory Systems
View Paper: https://www.semanticscholar.org/paper/66cd7c05da828bf80e3e965eb8dcbe35addfa65b
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
- Z. Xiong
- Xiao-man Zhu
- J. Macdonald
- Jessica Hey
- Roger Simon
- Wenlin Wei
- X. Chu
- M. Minami
Citations in the corpus
- Cellular and Molecular Mechanisms of Pain (2009, neutral): “…mice display only a slight decrease in action potential firing to mechanical stimuli, whereas ASIC3-deficient fibers display a slight increase (no change in mechanical thresholds or baseline behavioral mechanical sensitivity was observed in these animals) (Price et al., 2001; Roza et al., 2004).”
- Neuroprotection in ischemia: blocking calcium-permeable acid-sensing ion channels. (2004, background): “In Results peripheral sensory neurons, ASICs have been implicated in mechanosensation (Price et al., 2000, 2001) and Acidosis Activates ASICs in Mouse Cortical Neuronsperception of pain during tissue acidosis (Bevan and Yeats, 1991; Krishtal and Pidoplichko, 1981; Ugawa et We first recorded ASIC…”
- TRPM8 is required for cold sensation in mice. (2007, neutral): “However, gene ablation studies of TREK-1 and DEG channels in mice have not yet pointed to required roles in cold sensation (Alloui et al., 2006; Price et al., 2000, 2001 ).”