Nervous system reorganization following injury.
By Robert Chen, Leonardo G. Cohen, Mark Hallett. Neuroscience, 2002
736 citations recorded by the source corpus.
Subfields: Neurology, Rehabilitation
View Paper: https://www.semanticscholar.org/paper/66a17a3ed8045278d677edbd9c584ed749a2ac14
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In Neurology
In Rehabilitation
Citations in the corpus
- Is the mind really modular (2005, background): “This conjecture is supported by the plasticity of the senses (Chen et al. 2002).”
- White matter maturation reshapes structural connectivity in the late developing human brain (2010, background): “However, animal and human experiments have shown that white matter rewiring through the significant growth of new axonal pathways or axonal pruning is unlikely to occur in late development, particularly in the absence of a major injury (21, 22).”
- Hand function: peripheral and central constraints on performance. (2004, background): “Such a widely distributed network of diverse elements would provide a flexible substrate for practice-driven plastic reorganization, advantageous both for the motor skill learning needed to play a musical instrument and for functional recovery after nervous system injury (11, 59, 67, 80).”
- Repairing the human brain after stroke: I. Mechanisms of spontaneous recovery (2008, background): “However, note that persistent, increased reorganization is not always helpful because persistence of poststroke plasticity can be associated with induction of epilepsy(157) or chronic pain.(158) In some respects, these patterns of change recapitulate an adaptive pattern seen in the healthy state.”
- Cell type-specific structural plasticity of axonal branches and boutons in the adult neocortex. (2006, positive): “Moreover, structural plasticity may be critical for functional recovery upon injury in the adult central nervous system (Fawcett and Geller, 1998; Chen et al., 2002; Papadopoulos, 2002).”
- Neural plasticity after peripheral nerve injury and regeneration. (2007, background): “Removal of inhibition of excitatory synapses due to reduction of GABAergic inhibition has been suggested as the most important to cause short-term plastic changes (Chen et al., 2002).”
- Harnessing neuroplasticity for clinical applications (2011, neutral): “The participants identified cardinal examples of human neuroplasticity in these conditions, underlying biological substrates and mechanisms, promising interventions for promoting adaptive neuroplastic changes and measures for evaluating neuroplastic capacity and monitoring circuit engagement and…”
- A network dysfunction perspective on neurodegenerative diseases (2006, background): “This concept is less radical than it might seem at first glance, given that effective neural plasticity allows the brain to cope surprisingly well with even striking neuronal losse”